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As an antioxidant, selenoprotein M and selenoprotein W-like have been reported that involved in alleviating the oxidative stress and apoptosis caused by virus infection (92, 93)

Mechanisms of eNOS uncoupling Under physiological conditions, i.e

Although challenges exist due to its solubility and cytotoxicity, advancements in DDS have shown promise in improving its therapeutic application

We observed that induction of R175H expression in OVCAR5 cells resulted in a more significant reduction in cell proliferation upon treatment with APR-246 (Fig