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With respect to targeted therapeutic strategies, the focus on key transporters and metabolizing enzymes (ASCT2/GLS1) provides a theoretical foundation for the development of multi-targeted combination therapeutic regimens based on the inhibition of glutamine metabolism

Interestingly, some studies have shown a relationship between structural abnormalities of myelin in ADs in the pre-clinical period and peptide concentrations of A142 in patients CSF [34, 38]

Several of these have progressed to phase 3 clinical trials which are either ongoing or have failed their primary outcomes

Here the review turns to precursor support nutrients that dont replace glutathione, but help the body rebuild it from within