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Failed heart muscle is metaphorically referred to as a fuel-depleted engine. If mitochondria are impaired in their ability to convert energy substrates into fuel (ATP), the burden on heart function increases

Based upon drug down selection data and measured EC 50 values, Phenylbutazone, Enoxacin, and Doripenem hydrate were selected for in vivo validation in mice with H2AX foci per nucleus IHC staining as an outcome measure consistent with prior work showing correlation with the development of xerostomia 17, 41, 42, 46