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In this cycle, GSH is released from the cell by carrier mediated transporter(s) and the ecto-enzyme GGT then transfers the -glutamyl moiety of GSH to an amino acid (the best acceptor being cystine), forming -glutamyl amino acid and cysteinylglycine

doi: 10.3390/nu12113567 246

Additionally, it was reported that trimetazidine (TMZ), a piperazine derivative, mediated activation of the GSH system and had antioxidant and cytoprotective effects in several myocardial injury models [210,211,212,213]

Compounds such as NCX4040 deplete intracellular GSH and elevate lipid peroxidation markers, and their effects are significantly enhanced when combined with ferroptosis inducers such as erastin or RSL3 (121)