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In the presence of GSH, hemolytic activity of LLO in vitro was eliminated [38]

227 In pathological iron overload, this DMT1 iron transport process is aggravated by increased TFR1 saturation, leading to the generation of toxic NTBI, which then causes cell damage through ferroptosis

Inflammatory cytokines, which are known to activate ER stress pathways, have been shown to induce both citrullination and deamidation of beta-cell proteins (79, 80), such as GRP78 and proinsulin