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Promising therapeutic effect of thapsigargin nanoparticles on chronic kidney disease through the activation of Nrf2 and FoxO1

After TBI, there is an activation of neuroinflammation response pathway mediated by microglia cells that were not initially damaged, and the activated microglial cells may promote the release of proinflammatory factors such as interleukin 1, tumor necrosis factor, nitric oxide, etc., (Kumar and Loane, 2012), inducing a progressive cycle of chronic inflammation (Sulhan et al., 2020)

Compound Information Technical Specifications Sources & References Peer-Reviewed Research Biomolecules 2018 Regenerative and Protective Actions of the GHK-Cu Peptide in the Light of the New Gene Data View Source Journal of Investigative Dermatology 1985 The stimulation of protein synthesis by the tripeptide-copper complex glycyl-l-histidyl-l-lysine-Cu(II) View Source PLoS ONE 2012 GHK-Cu may prevent oxidative stress in skin View Source Wound Repair and Regeneration 2004 The human tri-peptide GHK and tissue remodeling View Source For Research Use Only Important Notice This product is sold strictly for research, laboratory, and educational purposes only

D.LinK.ZhengG.ZouH.et al (2015)