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130 Cancer cells balance ROS levels to promote redox signaling and survival while avoiding oxidative stress, a vulnerability that therapies targeting these pathways aim to exploit, selectively inducing oxidative damage and cell death in tumors (Fig

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Furthermore, UGTs are involved in the regulation of several active endogenous compounds such as bile acids or hydroxysteroids due to their inactivation via glucuronidation (Miners & McMackenzie, 1991, Kiang et al., 2005)