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Finally, we emphasize a critical knowledge gap regarding the role of the tissue environment as a modifier of glucocorticoid sensitivity and stress the need for integrated omics approaches to uncover tissue-specific epigenetic modifications associated with perinatal disease and clinical response to glucocorticoids

Acrylamides can covalently bind to cysteine clusters on target proteins, making them commonly referenced as electrophilic groups in the structural design of covalent inhibitors

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Although it is easy to overwhelm this protective mechanism in first-time patients receiving cisplatin, continues exposure to the drug ultimately produces resistance due to increased sulfhydryl levels (e.g., GSH and MT) (Takehiko et al ., 2001