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One important mechanism behind endothelial cell dysfunction in ARDS is ferroptosis, a kind of cell death caused by iron-dependent lipid peroxidation ( Figure 4 )

Collectively, these results indicate that oxidative stress is a powerful driving force behind antiretroviral-induced toxicity and has important roles in premature-and-accelerated-aging symptoms (Blas-Garcia et al., 2011)

Siddiqui, Virginia Commonwealth University, Michael Silver, Labcorp, Margery Connelly, University of Florida and Arun Sanyal, Division of Gastroenterology, Hepatology, and Nutrition, Virginia Commonwealth University, Richmond, VA Background: Liver plays a central role in lipoprotein metabolism and is a key driver of atherogenesis

These findings highlight the crosstalk between ferroptosis and apoptosis, encouraging further research to elucidate the regulatory mechanisms of the ferroptosis-apoptosis pathways