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Although the exact pathophysiology is unknown, this phenomenon could result from cranial nerve VII inflammation and edema caused by viral infections (219)

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Drugs 77 , 17051711 (2017)

The accumulation of misfolded protein in the endoplasmic reticulum (ER) leads to ER dysfunction, which is known as ER stress ( 12 regulated ER homeostasis ( 12 deficiency activated ER stress pathways by increasing the phosphorylation of PERK and IRE1 and the expression of ATF6 ( 12 could promote nerve regeneration after TBI by regulating ER stress