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We further found that the glutathione metabolism pathway was one of the most significantly affected pathways, with some key functional genes up-regulated (Wang et al., 2019a), no burst of oxygen free radicals was detected, and the activity of various antioxidant enzymes including glutathione reductase (GR) was up-regulated in B

The intersection of DNA damage response and ferroptosisa rationale for combination therapeutics

However, the distinctiveness of PEM lies in the manner in which this reprogramming is triggered and its self-amplifying nature: it is not directly induced by pathogens but is instead initiated by a potential energy crisis resulting from mitochondrial dysfunction

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