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Treatment of the proteins with DTT prior to application to the array allowed us to distinguish between those proteins where BioGEE had been incorporated into the protein via a non-disulphide mechanism (possibly due to interaction with biotin) from those that formed disulphide bonds with the GSH in BioGEE and hence were more likely to represent potential targets of glutathionylation

this indicates that bromfenvinfos caused cell shrinkage and phospholipid mixing in the erythrocyte cell membrane, which is partly due to stimulation of ROS formation and Ca 2+ entry [437]

Autophagy 10 , 13691379 (2014)

In published literature the individual components appear in models of connective-tissue repair, angiogenesis, cell migration, and copper-dependent extracellular-matrix signaling