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Disruptions in regulatory mechanisms lead to the excessive proliferation and abnormal arrangement of collagen fibers, resulting in lesions that exceed the scope of normal physiological repair and are accompanied by functional impairments or cosmetic abnormalities (24)

This transition is due to sII opening and the distortion of intramolecular interactions causing the breaking of salt bridges and making the subsequent oxygen binding process less demanding (34, 35, 56)

Supplementary material The Supplementary Material for this article can be found online at: References 1 AkramW.KhanI.RehmanA.MunirB.GuoJ.LiG

Q3ME suppressed esophageal cancer cell proliferation as well as the malignant transformation of healthy esophageal cells by inhibiting the MAPK and AKT/mTOR/p70S6K signaling pathways