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They maintain an equilibrium between astrocytic biosynthesis and oxidative degradation, which perhaps also is astrocytic and is capable of establishing net synthesis or net degradation according to glutamate needs

This approach mirrors how longevity-focused clinicians use other interventions, prioritizing a sustained, low-amplitude signal over a short, high-amplitude intervention

These results demonstrate that GSTA1 expression is negatively associated with the accumulation of LD and might be a protective factor for the progression of MASLD