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discontinue treatment if the patient cannot tolerate this maintenance dose

Collectively, these varied findings concerning different amino acids underscore the complex relationship between amino acid metabolism and atherosclerosis

Hence, the presence of l-alanine in the l-alanyl-l-glutamine dipeptide or in its free form and supplemented in tandem, can spare glutamine metabolism in order for the latter to be used by high-demand tissues under inflammatory and/or highly catabolic situations (Reference Cruzat, Pantaleao and Donato7,Reference Cruzat and Tirapegui32)

Among the identified gallic acid, protocatechuic acid, epigallocatechin gallate, caffeic acid, naringin, resveratrol, kaempferol, and biochanin-A are highlighted as the compounds responsible for the enzyme inhibitory activity (58)