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Third, lack of HO-1 expression in bone marrow-derived cells resulted in atherosclerotic plaques with a larger inflammatory component, suggestive of greater vulnerability for rupture (Orozco et al., 2007)

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Cardiac, renal, and cerebral malformations were consistent in fetal and neonatal ultrasound scans of both siblings, who had biochemical and molecular diagnoses, confirming CPT II deficiency

The root meristemless1/cadmium sensitive2 gene defines a glutathione-dependent pathway involved in initiation and maintenance of cell division during postembryonic root development