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As with other antimuscarinic agents, the major action of atropine is a competitive or surmountable antagonism which can be overcome by increasing the concentration of acetylcholine at receptor sites of the effector organ (e.g., by using anticholinesterase agents which inhibit the enzymatic destruction of acetylcholine)

All authors contributed to the study conception and design, commented on previous versions of the manuscript

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PRMT1, a key epigenetic modifying enzyme, is involved in various cellular processes, including gene transcriptional regulation, RNA metabolism, and cell signaling [46]