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Description

GSH can be directly oxidized by many intraluminal reactions involving the oxidative protein folding machinery

Overall, ATF1, ATF2, ATF4, ATF5 and ATF6 are persistently upregulated in most inflammatory, metabolic and stress-related diseases, acting primarily as transcriptional activators that drive pro-inflammatory programs or adaptive stress responses

These experimental results indicated that THJ can protect myocardial injury through antioxidative stress and anti-inflammatory effects, and the molecular mechanism of these effects may be related to the Sirtuin1/NLRP3 signaling pathway