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As healthcare providers continue to explore these options, the potential for personalized treatment plans that incorporate these innovative agents could revolutionize the way we approach these chronic conditions

Recent research indicates that neuronal ATP and adenosine interact with their respective receptors-P2X and P2Y for ATP, and adenosine A 2A receptors (A 2A R) for adenosine-leading to pericyte contraction, which reduces CBF, or membrane hyperpolarization, which increases CBF [26, 87,88,89]