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If that methionine residue plays a role in receptor binding or biological activity, oxidation can reduce or eliminate the peptide effect entirely

This helps protect cells from oxidative stress, supports detoxification pathways, promotes immune resilience, and contributes to overall cellular health

Furthermore, the administration of S-biAb/dEGCG@NPs not only strengthens IFN-mediated tumorcidal efficacy by targeting B7-H3 which prevents T cell activation, but also significantly downregulates GPX4, intensely boosting the capacity of immune checkpoint blockage therapy[175]

Protective effects of wogonin on lipopolysaccharide-induced inflammation and apoptosis of lung epithelial cells and its possible mechanisms