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Someone managing high levels of environmental stress or oxidative load may feel a more significant difference than someone whose levels are already well-supported by diet and lifestyle

Astroglial toxicity promotes synaptic degeneration in the thalamocortical circuit in frontotemporal dementia with GRN mutations

Shungu Once the process starts, it can be hard to tell what is causing what as the mitochondrial and oxidative stress problems feed into each other: In our study of primary mitochondrial disorders (e.g., MELAS), we have found a cortical GSH deficit that seems to be even bigger than in ME/CFS, meaning that mitochondrial dysfunction and oxidative stress co-exist and likely potentiate and sustain each other in such disorders

This stimulatory effect was also observed in primary cultures of human and mouse TEC when PRL (10(-7) to 10(-8) M) was applied to culture supernatants, thus suggesting that PRL could act directly on TEC