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Results showed that LDR (0.090.11 Gy) significantly increased deletion mutants in HPRT in peripheral blood cells, and that the mutation frequency declined with time following exposure

This paper systematically reviews the molecular mechanisms of disulfidptosis, elucidates its regulatory role in male cancer cellsincluding key regulatory genes and therapeutic target potentialand discusses its application value and challenges as a potential therapeutic target based on clinical research

Moreover, replacements of Lys105 resulted in a reciprocal increase of the K m app (GSSCys) values and decrease of the K m app (GSH) values (Fig