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Copper deficiency can result from impaired absorption (due to alkaline pH, use of antiacid drugs, or food complexes such as phytic acid or other minerals), conditions promoting copper loss (e.g., nephrosis), or total parenteral nutrition

In cellular models, GSH depletion increases neuronal death in striatal neurons expressing mHTT, reinforcing the role of GSH-dependent redox imbalance in HD progression (400)

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Additionally, vitamin E may not be biologically important as an AT, but rather as a signaling molecule and regulator of specific gene expressions ( Various structurally related derivatives of vitamin E have been developed, some of which have improved antioxidant effects with in vitro AT assays or animal models