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Dual-specificity phosphatase 26 protects against nonalcoholic fatty liver disease in mice through transforming growth factor beta-activated kinase 1 suppression

doi:10.3390/ijms22168677

Bn c th d dng mua sn phm cc n v i tc chnh hng, ca hng chuyn cung cp cc sn phm lm p

Furthermore, while enzymatic removal of the L585 residue decreased FcRn binding for fusion designs with WT albumin, this was not the case for the corresponding QMP-engineered variants