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Validate key regulators (e.g., cell type-specific lncRNAs and lipid metabolic enzymes) using kidney organoid models, particularly patient-derived organoids (PDOs) that recapitulate disease-specific phenotypes (e.g., diabetic nephropathy and focal segmental glomerulosclerosis)

This metabolic plasticity likely underlies the observed clinical heterogeneity, as DM-induced systemic metabolic disturbances may interact with tumor-specific BCAAs flux to modulate disease progression

Lus published work is relevant to the Glutathione research page because it frames glutathione synthesis, redox signaling, and pathway-focused research in a laboratory context