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Notably, the recognition of limited or partial EndoMT does not necessarily conflict with its potential functional relevance in PA stiffening, but simply suggests that this relevance may potentially relate more to the release of proliferative, hypertrophic, and profibrotic signalsi.e., mediators of processes that will ultimately promote PA stiffnessby partial EndoMT cells rather than to the actual generation of significant mesenchymal cell mass via this mechanism

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