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For this purpose, we will summarize: 1) The current evidence that acutely-induced oxidative stress is cholestatic, by describing the mechanisms underlying the hepatocyte secretory failure, including the disorganization of the actin cytoskeleton and its most noticeable consequences, the impairment of tight-junctional structures and the endocytic internalization of canalicular transporters relevant to bile formation

DNA repair is not involved in the cell resistance To investigate whether U138MG resistance to cisplatin was because of an enhanced DNA repair capacity, host-cell reactivation (HCR) assays were performed using a firefly luciferase gene reporter

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