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At the hormonal level, ketogenesis is strongly regulated by insulin, as it inhibits the breakdown of stored fats in adipocytes and promotes glucose uptake and utilization, leading to elevated levels of inhibitors of fatty acid oxidation and KBs production, such as succinyl-CoA and malonyl-CoA (Johnston and Alberti, 1982)

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Similarly, GPXs in the endoplasmic reticulum aid in protein folding and stress regulation, while apoplastic GPXs modulate extracellular ROS to activate defense signaling and systemic acquired resistance (SAR) ( Figure 9 ) (Tripathy and Oelmller, 2012)