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Uptake of extracellular asparagine prevents the death of glioblastoma cells in response to glutamine depletion by blocking the apoptotic function of a glutamine-induced endoplasmic reticulum stress marker protein, ATF4, and by increasing glutamate-ammonia ligase (GLUL)-mediated glutamine and protein synthesis 116,117

Furthermore, marked increase of ascorbic acid turnover was reported on consumption of estrogen containing medications [81]

These EPB@pAzF-amyloid conjugates served as a high-throughput screening platform to repurpose FDA-approved drugs, small molecules, and natural components with amyloid inhibition functions against different amyloid proteins

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