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We first assessed the ability of entrectinib to cross the bloodbrain barrier and found that systemic entrectinib (120 mg kg 1 , oral administration) reduced pharmacodynamic markers of TrkB signalling, including TrkB phosphorylation and downstream ERK phosphorylation in brain tissue (Extended Data Fig

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Tissue iron promotes wound repair via M2 macrophage polarization and the chemokine (C-C Motif) ligands 17 and 22