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Neuroinflammation, oxidative stress and the pathogenesis of Alzheimer's disease

GSH serves as a potent anti-pyroptotic regulator through two primary mechanisms: maintenance of cellular redox homeostasis and post-translational modification of inflammasome components via SSG (Fig

Thus, substituting lung tissue for PBMCs must be well considered as studied expression changes might be compartmentalized to the lung (32)

Keywords: pentose phosphate pathway, glycolysis, glucose 6-phosphate dehydrogenase, NADPH, metabolomics, oxidative stress, cancer, stem cells, hostpathogen interactions, metabolic engineering, inherited metabolic disease, parasitic protozoa, metabolism of infection I