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Protein modifications by acrolein induce ferroptosis Acrolein irreversibly modifies proteins by forming covalent adducts with nucleophilic residues, such as cysteine, histidine, and lysine, significantly altering their structure and function, and potentially leading to cell death 57

PMID: 31627309

Suicidal erythrocyte death triggered by cisplatin

These mitochondrial perturbations result in electron leakage from respiratory complexes, generating excessive superoxide radicals while simultaneously compromising energy availability