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Similarly, the restoration of neurotrophic support, whether through exogenous administration, gene therapy, or immune-mediated modulation, illustrates a growing sophistication in therapeutic design, although questions regarding long-term efficacy, immune tolerance, and BBB penetration remain unresolved

We identified the underlying molecular mechanism: T-DXd treatment triggers an increase in the levels of p53 and p21

In a study of 30 children with celiac disease, 80% had serum selenium levels below the normal range (Yuce 2004)

According to the latest research, the excess of FAO results in inadequate mitochondrial function, which can lead to impairment of the electron transport chain