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Similarly, paclitaxel affects synaptic plasticity by increasing presynaptic NMDA receptor (NMDAR) activity, especially that of GluN2A-containing subunits, in the spinal cord, thereby mediating neuropathic pain (Xu et al., 2022)

doi:10.1016/j.bbamcr.2016.01.023

Increased transcription of IL-6 and IL-1, mediated through the ERK and the p38 MAPK signal pathways, occurs due to the combined effects of DON and PCV2, although similar effects may be exerted by PCV2 through the JNK signal pathway (115)