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However, a possible confounding factor is that ribose models tend to use a supraphysiological ribose concentration, which may induce an excessive AGE crosslinking profile

Widening the therapeutic window for redox-targeting drugs Given that SCLC cells are unable to adapt to redox-targeting drugs, such as TXNRD1 inhibitors, we hypothesized that activating the NRF2 pathway would enhance antioxidant defenses specifically in non-cancerous cells, thereby further widening the therapeutic window

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