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Abstract Atherosclerosis (AS) is a common cardiovascular disease with complex pathogenesis, in which multiple pathways and their interweaving regulatory mechanism remain unclear

In preclinical studies, telaglenastat (CB-839), a Gln synthetase inhibitor, promoted the function of CD4 + Th 1 cells and CD8 + T cells, and inhibited the function of Th 17 cells

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These findings indicate that limiting serine metabolism may have significant implications for immunotherapy, highlighting the importance of targeting these pathways in cancer therapy