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Though we understand some of the molecular mechanisms that regulate phenotypic switching of VSMCs, and a positive, negative or neutral effect of particular VSMC phenotypes on atherosclerosis and plaque stability, we currently lack experimental evidence in vivo from animal research or correlated data in human studies

The normal concentration of glutamate is necessary for the physiological function of the brain, but when it exceeds normal, it will cause the death of excitatory neurons and produce neurotoxic or excitotoxic events (Dong et al., 2009)

TBK1 (TANK-binding kinase 1)-mediated regulation of autophagy in health and disease

Antigen presenting cells (APCs)