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CJC-1295 Safety Concerns CJC-1295 has more direct human pharmacologic data demonstrating its effect on growth hormone and IGF-1

IGF-1 prevents ANG II-induced skeletal muscle atrophy via Akt- and Foxo-dependent inhibition of the ubiquitin ligase atrogin-1 expression

It accelerates tendon fibroblast outgrowth, increases cell survival under stress, and promotes cell migration, likely through the activation of the FAK-paxillin pathway