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An open therapeutic observation study of 2,512 patients]

To further separate transcriptional regulation from post-transcriptional, we conducted the cycloheximide (CHX) chase assay and found that PIN1 KO markedly reduced the half-lives of mitotic proteins (Fig

Genetic Predispositions and Antioxidant Pathway Efficiency Individual genetic variation in glutathione synthesis and metabolism enzymes influences baseline antioxidant capacity

Genetic or pharmacological blockade of CerS5 lowers C16-ceramide, silences Pla2g7NLRP3 signalling and markedly improves recovery, positioning CerS5 as a metabolic accelerator of the pyroptotic response (Zhang et al., 2025)