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When intra-portal insulin levels are increased (e.g., obesity, Cushings syndrome, or due to treatment with glucocorticoids and glucagon-like peptide 1 receptor agonists) or decreased (e.g., malnutrition, anorexia nervosa and type 1 diabetes mellitus), these changes secondarily alter hepatic GH sensitivity resulting in a secondary association with discordant GH and IGF-I levels (e.g., high GH/low IGF-I levels or low GH/high IGF-I levels, respectively)

Some research suggests immunomodulatory effects with potential to enhance immune functions that decline with aging

In addition to affecting, regulating and inducing sleep, it also has other remarkable effects, for example, on the circadian rhythm

Insulin resistance and growth retardation in mice lacking insulin receptor substrate-1