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We employed diamide, an established thiol-oxidizing agent that oxidizes GSH to GS-SG [22], to induce GSR-mediated NADPH consumption during reduction of GS-SG back to GSH (Fig 1A)

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[] Consequently, the focus has shifted toward upstream targets, such as reducing oxidative damage with mitochondrial-targeted antioxidants and fine-tuning the mitophagy of damaged organelles

The severity and extent of these infections depend on several factors, with the hosts weakened immune system being the most significant one [11]