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In Bcl-2+ cells (MDCK introduced by Bcl-2), p38MAPK interacts with Bcl-2 and colocalizes in the cytoplasm, and both Bcl-2 phosphorylation and apoptosis are decreased by specific p38MAPK activity

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3.3 Targeting VEGF-mediated tumor angiogenesis Vascular endothelial growth factor (VEGF) drives tumor angiogenesis through hypoxia-inducible factor 1 (HIF-1)-mediated transcriptional regulation, a process amplified by oncogene signals, growth factors, and hypoxic stress