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Because of the complex, emerging psychosocial and neurodevelopmental needs of patients with ASD, pharmacological management of autism-associated behaviors is a must for APRNs (1,2,3,4,5,6)

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In addition to modulating the posttranslational modifications discussed above, improving metal delivery to SOD1 is reported to yield the greatest therapeutic benefit to multiple strains of mutant SOD1 transgenic mice to date.[ 151 , 154 , 228 ] This is consistent with data demonstrating that the toxicity of misfolded mutant SOD1 protein is more closely associated with SOD1 metal content than the total amount of mutant SOD1 protein in these mice