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This challenge was addressed by replacing disulfide linkages with 3-sulfanylpropionyl ester linkages that are sensitive to the acidic pH of endosomes

Elevated levels usually indicate pulmonary issues

Our data indicated that ALDH3A1 positively regulated the Nrf2/HO-1/GPX4 axis to suppress ferroptosis and functioned as a critical downstream target of AHR signaling