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demonstrated that S-nitrosothiols are essential in NO biology and influence blood pressure (24), and it has also been reported that thiol supplementation with GSH selectively improves human endothelial dysfunction by improving NO bioavailability (29)

Depletion of GSH due to intensified oxidative stress or the action of inhibitors significantly reduces the ability of GPX4 to counteract lipid peroxidation, leading to increased lipid peroxides and subsequent ferroptosis (151)