glutathione synthetase deficiency schizophrenia

glutathione synthetase deficiency schizophrenia Glial cell deficits are a key feature of schizophrenia: implications for neuronal circuit maintenance and histological differentiation from classical neurodegeneration Nineteen-year follow-up of a patient

Name: glutathione synthetase deficiency schizophrenia Glial cell deficits are a key feature of schizophrenia: implications for neuronal circuit maintenance and histological differentiation from classical neurodegeneration Nineteen-year follow-up of a patient
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Nineteen year follow up of a patient with severe glutathione synthetase deficiency Journal of Human Genetics Schizophrenia and Glutathione: A Challenging Story Mitochondrial dysfunction in schizophrenia: evidence for compromised brain metabolism and oxidative stress Molecular Psychiatry Glutamate in schizophrenia: Neurodevelopmental perspectives and drug development ScienceDirect

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According to the results of the Rosetta Cartesian_ddG and Flex_ddG, we manually checked the PV2 and PV3 variants predicted to be highly stable, exhibit low binding free energy and are located outside the CDR regions, and those having pAcF facing outward were selected for experimental characterization, including PV2 variants S8pAcF, S26pAcF, S71pAcF, S74pAcF, Q82pAcF, S85pAcF and PV3 variants S8pAcF, S26pAcF, S71pAcF, N77pAcF, S85pAcF, K87pAcF (Fig