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Subunit-specific organization of NMDARs within spine synapses The synaptic content of GluN2A and GluN2B NMDAR subunits changes the ability of a synapse to undergo structural plasticity, suggesting that the specific ratio of GluN2A to GluN2B in NMDAR heterotetramers may tune synaptic function 1,9,10

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Folate deficiency could also intensify or progress symptoms of AD through hypomethylation

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