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Studies have found that Htr4 gene expression is suppressed in the hippocampus of AD patients, and activating 5-HT4 receptors can improve cognitive function [60, 61]

In rabbit studies with 0.8 cm radial bone defects that remained incompletely healed in all control animals for 6 weeks, BPC-157 treatment (administered locally into the bone defect or intramuscularly) produced healing comparable to standard treatments including local bone marrow application or autologous cortical bone grafting