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To identify pathways that were altered by vB12 treatment, we performed a pathway analysis (using a DAVID functional annotation tool) of up-regulated and down-regulated genes separately for each of the three categories of DEGs

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showed that the transcription level of Wnt3a in GMSCs was high, and the CD39-specific inhibitor POM-1 inhibited the expression of Wnt3a and reduced the level of -catenin, ultimately leading to loss of the osteogenic ability of GMSCs

In the case of macular degeneration, unfortunately, adhesions and communication between RPE cells are lost because of inflammation