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Iron dyshomeostasis in glial cellseven in the absence of cell deathcontributes to the vicious cycle: iron-loaded microglia directly secrete IL-1 and IL-18 to fuel neuroinflammation (Ryan et al., 2023), while reactive astrocytes release hepcidin and pro-inflammatory cytokines, which elevate neuronal iron load and sensitize neurons to ferroptosis (Xiong et al., 2016

Free Radic Biol Med 110:1118 Jiao X, Meng T, Zhai Y, Zhao L, Luo W, Liu P, Qin Y (2021) Ovarian reserve markers in premature ovarian insufficiency: within different clinical stages and different etiologies

Curry 1 , (1)Beth Israel Deaconess Medical Center, Harvard Medical School, Boston, MA, (2)University of Iowa Hospitals and Clinics, (3)University of Texas Southwestern Medical Center, (4)Mallinckrodt Pharmaceuticals, Bridgewater, NJ Background: Hepatorenal syndrome type 1 (HRS-1) is a rapidly progressive form of renal failure associated with high mortality in patients (pts) with decompensated cirrhosis and ascites

The 2006-2007 Phase 2 lipodystrophy trial was halted after a participant died